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FTX606 CV and ANS Cheat Sheet (DRAFT) by

Cardiovascular and Autonomic Nervous System

This is a draft cheat sheet. It is a work in progress and is not finished yet.

Cardio­vas­cular Effects of Sympat­hom­imetics

Barore­ceptor Reflex (General)

The Barore­ceptor Reflex is the rapid response system for dealing with BP changes (EITHER increase OR decrease).

1. If the BP is DECREASED, the baroreflex will initiate responses to INCREASE CO, causing vasoCO­NST­RIC­TION. End result: INCREASE BP

2. If the BP is INCREASED, the baroreflex will initiate responses to DECREASE CO, causing vasoDI­LATION. End result: DECREASE BP

The Barore­ceptor Reflex (DO and DO NOT)

Barore­ceptor DO
(1) Response to BP change. (2) Dampen any BP changes. (3) Dampen HR changes, revers­e/e­nhance HR changes.
Barore­ceptor DO NOT
(1) NO Response to HR change. (2) NEVER alter direction of BP change
 

General Hemody­namics

General Equation
Blood Pressure = Cardiac Output x Total Peripheral Resistance
Blood Pressure (BP)
Combin­ation of Systolic & Diastolic BP
Cardiac Output (CO)
CO = Stroke volume x Heart Rate
Total Peripheral Resistance (TPR)
alpha 1 receptor ALWAYS DOMINATE ("alpha domina­tes­")
Integr­ation:
Purpose: Maintain homeos­tasis & coordinate autonomic state by integr­ating afferent and efferent components of the SYMpat­hetic & PARAsy­mpa­thetic ANS within CNS

Direct Effects of Activating ANS receptors

(Note: ANS receptors are ranked based on relative effect)

1. Alpha1 receptors = MAJOR player = BIGGEST action.
2. Muscar­inic-2 receptors (vagus) works AGAINST Alpha1 receptor
3. Beta2 & Muscarinic (M, vascular) receptors are NOT attached (inner­vated) to Barore­ceptor Reflex.

Para- & Sympat­hetic Tone on BP & HR

Heart Rate: Vagus nerve (-10 bpm) + Beta1-­rec­eptor (+5 bpm) = 5 bpm increase

Influence of BP change on ANS tone