Decrease in Cardiac Output |
CAUSE: Heart muscle weakens or becomes stiff, impairing it's ability to contract & relax properly. |
Prevent the progression of the disease & decrease the risk of complications |
GOAL: Early recognition & management of decreased CO improves patient outcomes & quality of life. |
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INTERVENTIONS |
RATIONAL |
1. Auscultate apical pulse & assess HR |
- Body's 1st defense to compensate for reduced CO |
Objective Tachycardia = early sign of HF |
Persistent tachycardia is harmful & may worsen HF |
2. Obtain a comprehensive health history focusing on HF symptoms & self-management strategies |
Understanding patient's health history helps ID S/S of worsening HF |
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Also IDs patient's understanding and adherence to self-management strategies |
3. Note heart sounds |
An extra heart sound is caused by a large volume of fluid entering ventricle at the beginning of diastole |
Objective S3 (ventricular gallop) |
Indicates worsening HF |
4. Assess rhythm & document dysrhythmias |
A-Fib is common & promotes thrombus formation within the atria |
Subjective "Patient reports fast HR" |
Occurrence increases with HF severity |
5. Assess for palpitations or fast HR |
Palpitations occur due to dysrhythmias. |
Subjective "Patient reports fast HR, ""flutter" feeling" |
Fast HR may be compensation mechanism trying to get more blood flow back to the heart |
6. Palpate peripheral pulses |
Decreased CO may diminish radial, popliteal, dorsalis pedis, and post-tibial pulses. |
Objective Decreased pulse volume, cool, pale or cyanotic skin = decreased CO |
Evaluation helps determine adequacy of peripheral perfusion |
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7. Monitor BP |
Chronic HF, BP is used as a parameter to determine the adequacy or excess dosage of meds (ACEi) |
8. Inspect the skin for mottling |
R/T decreased perfusion to the skin |
Objectiveblue/grey skin coloring |
In chronic HF increased capillary oxygen extraction, skin appears dusky |
9. Inspect skin for pallor or cyanosis |
R/T to diminished perfusion |
Objective Cool, clammy skin |
10. Monitor urine output, noting decreasing output & concentrated urine |
R/T decreased renal perfusion |
Subjective Oliguria |
Fluid shifts into tissues during the day |
Subjective Nocturia |
Increased renal perfusion during supine position |
11. Examine LE for edema and rate it's severity |
Helps evaluate fluid status & guide diuretic therapy & fluid management |
Objective Pitting Edema |
12. Assess the abdomen for tenderness, hepatomegaly, and ascites |
Provides info on potential complications, guides interventions & Tx |
13. Assess jugular vein distention |
Estimates central venous pressure & IDs RV failure. |
Objective Distention greater than 4 cm |
14. Monitor Labs & Diagnostics |
Goal in diagnosis is to find the underlying cause of HF & patient's response to Tx |
15. Monitor O2 sats & ABGs |
Useful in establishing Dx & severity of HF |
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Provides info regarding the heart's ability to perfuse distal tissues w/ O2 blood |
16. Give O2 as indicated by the patient's symptoms, O2 sats, & ABGs |
Increases O2 availability to the myocardium |
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Helps relieve symptoms of hypoxemia, ischemia, & subsequent activity intolerance |
17. Provide a restful environment |
Minimizing controllable stressors & unnecessary disturbances reduces the cardiac workload |
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Providing physical & emotional rest allows patient to conserve energy |
18. Assist the patient into a High-Fowler's position |
Allows better chest expansion = improved pulmonary capacity |
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Reduced venous return to the heart |
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Relieves pulmonary congestion |
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Minimizes pressure on the diaphragm |
19. Check for calf tenderness, diminished pedal pulses, swelling, local redness or pallor of extremity |
Prolonged sedentary position increases the risk of thrombophlebitis, reduces CO, and increases venous pooling |
20. Encourage activity as tolerated |
**Chronic HF patient's should aim for 30 mins of physical activity daily |
21. Monitor for S/S of: |
Fluid Imbalance |
Fluid shifts & diuretics can cause excessive diuresis, leading to HYPOKALEMIA |
Electrolyte Imbalances |
S/S HYPOKALEMIA: VTach, Hypotension, Gen. weakness |
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ACEi & ARBs can cause HYPERKALEMIA |
22. Monitor Tele monitor & CXR |
Can indicate underlying cause of HF |
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CXR: May show enlarged heart & pulmonary congestion |